āThe death count is false.ā
If I lost someone close to Covid-19, I wouldāve punched that guy. Just saying.
Nuanced research into why some school districts have experienced more academic loss during and after remote learning due to the pandemic (hint: high poverty areas donāt have consistent access to high quality remote learning, and are more likely to have suffered family loss as a result of the pandemic):
is it just me, or does that pufferfish look suspiciously like the covid virus?
three days later and i finally feel somewhat more human. approximately like having a slight cold. ( i can even look at my phone screen for more than 5 minutes without nausea. oh, and eating. i can eat somewhat now. thatās cool too. )
apparently symptoms from flu and covid ( and even rsv ) overlap so much itās impossible to know for sure without testing which i had/have*. itās been so unlike my prior flu experiences, iām guessing covid.
cannot recommend. and if this was āmildā - because i have had all my shots except the bivalent - yikes.
will call the health plan tomorrow to see what they think about getting tested safely.
( * apparently loss of smell is the big telltale difference. in my case everything smelled so strongly it was nauseating.
but that was okay, because i could barely even keep water down anyway. )
If itās Covid & you qualify for paxlovid- it has to be started within 5 days of symptoms beginning to get approved.
paxlovid worries me just because of the rebound stories. i work from home, and no one will really miss me if im not back in action right away. iād rather just slow train through it once, and know itās done
now a throat lozenge? i would have killed for a throat lozenge.
( only figuratively of course. because literally i could have tried shambling out to get some, and killed several people with my germs. itās disheartening to think people tend to be the most infectious the day before symptoms appearā¦ )
I think itās like 20% who rebound. I didnāt. They probably needed to be on it a day or two longer; but 5 days is what they give. 
One wonders just how long the Chinese government can keep this āZero Covidā policy in place. But they do seemed determined:
[This] discovery also has the potential to help us understand Long COVID. āThe predominant features of that condition ā fatigue and neurologic dysfunction ā could be due to the lingering effects of mitochondrial damage caused by SARS-CoV-2 infection,ā he explains.
The paper:
https://www.sciencedirect.com/science/article/pii/S2213231722002804?via%3Dihub
But, it adds more data to a puzzling phenomenon. Scientists still arenāt clear why rebounds occur, who is at risk, or how common it is. Some have speculated that it could be due to rebounding viral replication or faltering immune responses. Some preliminary data from the National Institutes of Health, however, suggested that rebounds may be due to immune responses flaring as the body clears debris from a quashed infection.
I havenāt said WDKS for a while, but truly, WDKS.
An update yesterday from Dr. Bettina Hohberger at Erlangen.
My goodness but she speaks terribly quicklyā¦ from my notes:
They have been building a the network of both research and clinical people for ME/CFS and Long COVID. The network includes scientists and clinicians from many disciplines, including virologists, biochemists, and pharmacologists.
Work continues on studies in lab and in the Erlangen clinic as well. The reCOVer project is finalizing their study protocols. They are looking forward having the Berlin Cures documents and approval from the regulators for the next BC-007 study.
In the lab they are continuing the research into how autoimmune antibodies work. They are looking into the exact mechanisms for how these antibodies work the the goal of understanding Long COVID better. Their goals are to improve and speed up diagnosis, understand the mechanisms of Long-COVID, and speed access to new therapies.
Right, though I guess I do know that Iām not going to have rebound symptoms if I keep being careful enough to not catch it in the first place. 
