I guess “extremely low” is relative.
Caution is warranted here. Having a cold may be protective while you have the cold. As soon as you get over it, your risk goes back to baseline. No long lasting immunity, only the “one at the trough at a time” rule which is fairly common in viral infections.
Why is that? Does a viral infection put the immune system on higher alert for other viruses or do the viruses compete with each other or what?
My concerns:
No, the virus that first invades the cell hijacks the same machinery that any subsequent viruses would need. Makes it much harder for 2 viruses that invade the same type of cell to coexist. Now, that is an important qualifier, “same type of cell.” Having hepatitis C is not protective, Covid does not invade liver cells. But cold viruses compete for the same host cells, and have been adapting to humans for eons longer, and so are much better at it. Covid is a relative piker in comparison.
That’s presumably the case here:
If rhinovirus and Sars-CoV-2 were released at the same time, only rhinovirus is successful. If rhinovirus had a 24-hour head start then Sars-CoV-2 does not get a look in. And even when Sars-CoV-2 had 24-hours to get started, rhinovirus boots it out.
“Sars-CoV-2 never takes off, it is heavily inhibited by rhinovirus,” Dr Pablo Murcia told BBC News.
But as soon as your immune system shakes off the cold, the welcome mat is back out for Covid-19.
Is it the sort of thing where you could get infected with both but Sars-CoV-2 just waits out the Rhinovirus and then it gets you? Does Sars-CoV-2 have a dormant phase like herpes or hiv? Do the long-haul infections involve a dormant virus?
The authors make it sound like it gets expunged, provided it hasn’t progressed too far (i.e. more than 24 hours). This is not being observed in live subjects, mind, just a (very complicated) petri dish.
That is a gift! I hope your zinc experiment results in less pain
Just 12 idiots. I am so tired of the smallest of small minority fucking up things for the rest of us. Shut them down.
For all those “But kids don’t die from it! Send them back to school!” types out there:
Inside the ‘cyclone’ of brain fog many COVID-19 long-haulers are still experiencing - ABC News (go.com)
A whole generation of kids for whom this is the norm. Does that sound good to you? I don’t like it one bit.
They’re not idiots, I assure you that they are making a figurative killing to go with the literal killing.
The variants are going to be the difference maker in this thing. If we get vaccinated faster than the damn thing can spit out mutants, we win. We are not winning thus far, though, and the antivaxxers are not helping any.
As terrified as I am of experiencing any of that firsthand, I can only assume that it’s even more harmful for the developing brain. Because at this point in my life, when I’m whatever combination of fatigued and ill that most closely mimics that state, I at least have decades of previously acquired and solidified knowledge and judgement to lean on. As well as the rather reassuring knowledge/memory of not having felt like that the previous week.
Can’t answer that with any certainty. It definitely makes sense. I fear that over the coming years we will get to know that answer with great certainty, for better or for worse.
The move illustrates the Hong Kong government’s sensitivity to any criticism of the Sinovac vaccine, which has a comparatively lower efficacy rate and was fast-tracked by regulators despite a lack of published data.
Data on the jab from trials in other countries points to efficacy of between 50 per cent to 80 per cent, depending on the studies, compared with Pfizer/BioNTech’s 94 per cent to 95 per cent.
Not even a mention of what is happening in Mexico with the virus.
Mexican lives < $
re. Sinovac - despite a lack of published data.
Speaking of anekdota … I’ve not found anything about the outcomes of the RadVac experiment. They omit anything that might look like a result on the web site (I get why…)
I’m looking for allegedly informed gossip…
